SHERIFFDOM OF GRAMPIAN, HIGHLAND AND ISLANDS
AT ABERDEEN
JUDGMENT in causa MRS MAUREEN ELSIE SMITH or BROWN Pursuer against STUART FORSYTH Defender and MOTOR INSURER'S BUREAU Minuter
A2279/99 |
The Sheriff, having resumed consideration of the cause found the following facts proved.
Accordingly, finds in fact that the pursuer sustained injury as a result of the fault and negligence of the defender on 20th February 1997 and is entitled to reparation therefor; that the sum of £2790.00 is a reasonable estimate of damages sustained by the pursuer, and therefore grants decree for the sum of £2790 with interest as craved and appoints parties to be heard on
Note:
This is a reparation action following upon a road accident in February of 1997. There is no appearance for the defender, but the Motor Insurer's Bureau have entered the process as minuters to oppose the quantum of damages being sought.
The dispute here is essentially a disagreement between medical experts on cause of injury.
The facts of the accident are not in dispute. A car skidded across the road in front of the pursuer's car causing a collision which resulted in her being thrown forward into her air-bag which had inflated and then back and then forward again. The collision generated a shock for the pursuer and she was also aware of pain in the neck and shoulders. She immediately visited her own GP, was referred to the Accident and Emergency Unit at Aberdeen Royal Infirmary where a general diagnosis of whiplash injury was made and she was issued with a collar and given advice on self-treatment and pain killers. The pain eventually settled and she was able to resume normal activities but, not surprisingly, with some circumspection and care. In particular, she was able to resume golf. She returned to competitive golf on 30th June and won the Bronze Division of the Banffshire Ladies Championship, thus providing yet further circumstantial evidence in support of the old adage "beware the injured golfer". (It would seem to me to be a not unreasonable inference that the awareness of the injury made her more self-aware and more careful, resulting in the improved performance.)
However, the following day she awoke with pain in her neck radiating down her left arm. This pain gradually got worse as the day wore on, and soon became excruciating. She reverted to using the collar with which she had been supplied in February, but to no effect and she sought medical advice and also went to see a sports physiotherapist. Neurological involvement was apparent to her general practitioner on the 4th July and on the 14th July a suspected prolapsed disc was recorded. This was eventually confirmed and the pursuer underwent, fortunately, very successful surgical intervention in September from which she, in due course, fully recovered. The real dispute in this case was whether the July incident was related to the February incident. There is polite but flat disagreement between the medical experts on the existence of a causal link between the two incidents. There is no doubt that the pursuer suffered a prolapsed intervertebral disc on the 1st July and the first question is whether she suffered prolapse of the disc in February which then settled, but recurred in July with much greater severity of symptoms. Alternatively, even if she did not suffer a prolapsed disc in February, was her neck so damaged that the likelihood of a prolapse occurring was increased?
Before dealing with the conflict of opinion between the medical experts, I require to resolve apparent contradiction on the facts.
Paraesthesia (colloquially known as 'pins and needles') is an indicator of neurological damage. In any injury involving vertebral structures, doctors routinely look for signs of neurological damage or other damage affecting nerve roots. The mechanical explanation is that damage to or near a disc causes it to bulge, press on the protective sheath that surrounds the nerve root, causing pressure on and stimulation of the nerve which serves other parts of the body, in this case the left arm. Pain caused by damage to muscles and surrounding tissues is of different character and is sensed locally. If there was Paraesthesia after the February event, that would indicate the likelihood that there had been prolapse of an intervertebral cervical disc.
The primary source of evidence is the pursuer herself. It was clear from her evidence that the type of pain in July was quite different from the type of pain in February, and there was clearly an indication of Paraesthesia and radiating pain in July whereas, in February she refers to the aching of neck and shoulders. She is also the primary source of evidence for the various doctors, Professor Rowley examining the pursuer some time later was aware of the medical opinions expressed which related the two events. He explained that he took a very careful history and it was clear from the pursuer that she did not recollect any Paraesthesia in February. He reports that she did not have or develop arm pain. Mr Matheson does not record or recollect any complaint of Paraesthesia following upon the road traffic accident. Mr Currie, the consultant neurosurgeon who was responsible for the later operation, subsequently wrote a report narrating a history after the road accident in which he states that she had pain in her left arm and altered sensation in the form of 'pins and needles'. In evidence, however, he explained that this history was derived from the case notes and not from the patient directly. The case notes include two references to Paraesthesia. On 28th February there is a note from a Dr Moira Collins, who refers to Paraesthesia down the inner aspect of her "good" arm and left fingertips. That, presumably, is a reference to her right arm. There is no other evidence to involvement of her right arm. The next reference in the records is on "discharge summary" by the physiotherapist in a report to the GP practice in which, under the heading 'Diagnosis', is included "RTA whiplash injury, Paraesthesia of inner aspect of left arm and pain of upper and medial border scapular region". As there was no further evidence from the physiotherapist before the court it is not clear whether that purported diagnosis is the product of examination by the physiotherapist or simply repetition of what appears in the referral note. It seemed to me clear from Mr Currie's report and his evidence that he was assuming that the reference to Paraesthesia related to the left arm and not to the right arm. Her GP, Dr Carroll, is directly involved in July and he notes the Paraesthesia at that stage only, thus the first reference to Paraesthesia following the road traffic accident is from Dr Collins, but that relates to the right arm and the left hand. The subsequent references are derived from the records and not from direct examination with the possible exception of the physiotherapist. From Mr Currie's evidence it is clear that his opinion is based on the existence of Paraesthesia following the road traffic accident and Paraesthesia following upon the golf incident. Interestingly, the Accident and Emergency report records there was no neurological deficit on the 21st of February. Accordingly, I do not find Mr Currie's evidence helpful in resolving the issue of whether or not there was signs of neurological damage in February. It appeared to me that the greater probability clearly favours the view that there was no neurological damage directly observed following upon the road traffic accident.
That, however, is not the end of the matter. Mr Matheson is strongly of the view that there is an inference available from the July event, that the pursuer was predisposed to a prolapse because of the injury in February. This is the source of the major disagreement between the medical experts.
There is, in my view, no scientific evidence to support the inference advanced by Mr Matheson. I do not find the argument "post hoc ergo propter hoc" a persuasive argument in any and certainly not in these circumstances. One of the main purposes of expert evidence is to provide a rational basis for conclusions such as a causal link between two incidents. All the experts are agreed that there is no information available which can be demonstrated to be likely to be a causal link. There is no evidence of damage to an intervertebral disc or its surrounding tissues from which it can be inferred that there is a weakness or a predisposition to further injury, or even different injury. While it is agreed that a trauma is often referred to in patient histories, there is no demonstrable consequence to the trauma which leads to the conclusion that the likelihood of subsequent prolapse is increased. It is, in my view, illogical to conclude that all incidents which are subsequent to a trauma are caused by that trauma. In the current state of medical knowledge it is not even possible to say that any prolapse of disc subsequent to trauma is caused by the trauma except in most extreme circumstances where other injuries are also patent.
Accordingly, I do not accept the inference relied on by Mr Matheson. I fully understand the temptation to associate the two incidents, given the relatively short time between them. There was no evidence from which it could be inferred that the activity on the golf course would be liable, per se, to cause a prolapse to a cervical disc. It seems to me it is for the experts to rationally justify the conclusion that the disc had been weakened and, as Mr Matheson put it, was "waiting for something to happen". Mr Matheson may be right, but the current state of medical science does not allow a demonstration of cause and effect. Professor Rowley explains that, although a massive amount of attention has been devoted to seeking to discern the effect of trauma on cervical discs, particularly in whiplash context, no detail has been found to demonstrate a weakness in the disc or surrounding tissues which is the premise on which Mr Matheson's conclusion was based. Professor Rowley dismisses the conclusion as a speculation on possibilities rather than a rational conclusion on balance of probabilities. Professor Rowley also explained that it was common, or as he put it, human nature, to associate successive traumatic events and link them. As the function of experts science, however, is to demonstrate the truth or falsity of that connection, there was no evidence to demonstrate of the truth of the connection in this case. I accept his view on this matter. It may be that Mr Matheson is right, but there is no basis on which I can reach a conclusion that it is proved on the balance of probabilities the two incidents are causally linked.
Accordingly, I am driven to the conclusion that the incident in July is not proved to be related to the incident in February and, accordingly, the loss suffered after July is not referable to the road accident.
I turn now to consider the question of damages. I fully accept that the pursuer suffered episodically excruciating pain which gradually settled. In general terms, the assessment by Mr Matheson that she suffered four weeks of severe pain and four weeks of mild pain was supported by her own evidence, and she was virtually fully recovered by May. I do not find that the fact that she only lost four days work in February yielded a conclusion that the pain was less severe than she said. It seemed to me entirely credible that she would regard her duty to the children in her care as paramount, and she would force herself to go to work notwithstanding her discomfort. For a fit and active person such as the pursuer, disablement and restriction of movement is particularly distressing. It was agreed that £40 was a reasonable estimate of the additional expense of the pursuer being chauffeured to school. I was referred to various cases on the question of general damages. I find no difficulty in accepting the submission that £2,500 is an appropriate figure. In addition, the pursuer is entitled to a sum assessed in terms of the application of section 8 of the Administration of Justice Act 1982 which I would consider reasonably assessed at £250.
If I were to be wrong in concluding that there was no causal connection between the car accident and the prolapse in July, I would require to assess damages for the pain and suffering sustained after 1st July.
The pain after 1st July was significantly more disabling. The persistence of pain generated sleeplessness and general emotional disturbance. The use of painkillers produced a dependency which required to be reversed. Surgical intervention was necessary under general anaesthetic and the pursuer was in hospital for just under one week. She recovered from a successful operation over the next four months. solatium in general for the period after 1st July would be reasonably assessed at £4,000. In addition there was wage loss at an agreed figure of £250 and travel costs of £112 and physiotherapy at £82. In addition a figure requires to be assessed in respect of solatium for loss of holiday and compensation for household services (as per Section 8 of Administration of Justice Act). I would assess each at about £500. Thus a total figure would be reasonably stated at £5,444 for the period after 1st July.
Motion was made at the conclusion of the evidence for certification of Messrs Currie and Matheson as expert witnesses. I see no difficulty in granting that certification, but I shall put the whole issue of expenses out to a separate hearing.